Nursing practice questions with comprehensive rationales
NurseDive Free Nursing Practice Question
A nurse is preparing to discharge an obese client after abdominal surgery. Which instruction should the nurse prioritize to promote wound healing and prevent complications?
A. You should avoid taking pain medication to prevent constipation
Avoiding pain medication may increase discomfort, reducing mobility and increasing complications like atelectasis. Opioids can cause constipation, but this is managed with stool softeners, not avoidance. This statement is inaccurate, as pain control is essential for recovery and does not directly support wound healing.
B. You should rest and avoid moving around too much to prevent strain on your incision
Excessive rest increases risks like venous thromboembolism or pneumonia, especially in obese patients. Early ambulation promotes circulation and wound healing without straining incisions if done cautiously. This statement is inaccurate, as limiting movement excessively hinders recovery and increases postoperative complications.
C. You may return to your normal activity level once you feel comfortable at home
Returning to normal activity too soon risks incision dehiscence or infection, particularly in obese patients with higher wound complication rates. Gradual activity increase under medical guidance is needed. This statement is inaccurate, as premature activity resumption can compromise healing and safety post-surgery.
D. It is important to maintain adequate nutrition, including a high-protein diet to support wound healing
Adequate nutrition, especially high-protein intake, supports collagen synthesis and tissue repair, critical for wound healing. Obesity increases metabolic demand, and protein prevents malnutrition, enhancing immune response and incision strength. This statement is accurate, as nutrition is a priority to optimize postoperative recovery and prevent complications.
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Full Explanation
Choice A reason: Avoiding pain medication may increase discomfort, reducing mobility and increasing complications like atelectasis. Opioids can cause constipation, but this is managed with stool softeners, not avoidance. This statement is inaccurate, as pain control is essential for recovery and does not directly support wound healing.
Choice B reason: Excessive rest increases risks like venous thromboembolism or pneumonia, especially in obese patients. Early ambulation promotes circulation and wound healing without straining incisions if done cautiously. This statement is inaccurate, as limiting movement excessively hinders recovery and increases postoperative complications.
Choice C reason: Returning to normal activity too soon risks incision dehiscence or infection, particularly in obese patients with higher wound complication rates. Gradual activity increase under medical guidance is needed. This statement is inaccurate, as premature activity resumption can compromise healing and safety post-surgery.
Choice D reason: Adequate nutrition, especially high-protein intake, supports collagen synthesis and tissue repair, critical for wound healing. Obesity increases metabolic demand, and protein prevents malnutrition, enhancing immune response and incision strength. This statement is accurate, as nutrition is a priority to optimize postoperative recovery and prevent complications.
Similar Questions
Which statement best describes how physiologic doses of glucocorticoids are used?
A. Physiologic doses of glucocorticoids balance the feedback loop in clients with adrenal insufficiency
Physiologic doses of glucocorticoids, like hydrocortisone, mimic normal cortisol production (20-30 mg/day) in adrenal insufficiency, restoring hypothalamic-pituitary-adrenal axis feedback. This maintains metabolism, stress response, and immune function without excess. This statement is accurate, as these doses replace deficient cortisol to stabilize endocrine function.
B. Physiologic doses of glucocorticoids have the greatest impact on fluid and electrolyte balance
Physiologic doses have minimal impact on fluid and electrolyte balance compared to pharmacologic doses, which cause sodium retention via mineralocorticoid effects. In adrenal insufficiency, physiologic doses normalize cortisol without significant fluid shifts. This statement is inaccurate, as electrolyte effects are secondary and less pronounced.
C. Physiologic doses of glucocorticoids treat inflammatory disease in the body like rheumatoid arthritis
Physiologic doses replace cortisol in adrenal insufficiency, not treat inflammation. Pharmacologic (higher) doses suppress inflammation in diseases like rheumatoid arthritis by inhibiting cytokine production. This statement is inaccurate, as physiologic doses are insufficient for anti-inflammatory effects required in such conditions.
D. Physiologic doses of glucocorticoids lower blood glucose in place of insulin
Glucocorticoids increase, not lower, blood glucose by promoting gluconeogenesis and insulin resistance. Physiologic doses maintain normal glucose metabolism in adrenal insufficiency but do not replace insulin’s role. This statement is inaccurate, as glucocorticoids oppose insulin’s glucose-lowering effects, even at physiologic levels.
Full Explanation
Choice A reason: Physiologic doses of glucocorticoids, like hydrocortisone, mimic normal cortisol production (20-30 mg/day) in adrenal insufficiency, restoring hypothalamic-pituitary-adrenal axis feedback. This maintains metabolism, stress response, and immune function without excess. This statement is accurate, as these doses replace deficient cortisol to stabilize endocrine function.
Choice B reason: Physiologic doses have minimal impact on fluid and electrolyte balance compared to pharmacologic doses, which cause sodium retention via mineralocorticoid effects. In adrenal insufficiency, physiologic doses normalize cortisol without significant fluid shifts. This statement is inaccurate, as electrolyte effects are secondary and less pronounced.
Choice C reason: Physiologic doses replace cortisol in adrenal insufficiency, not treat inflammation. Pharmacologic (higher) doses suppress inflammation in diseases like rheumatoid arthritis by inhibiting cytokine production. This statement is inaccurate, as physiologic doses are insufficient for anti-inflammatory effects required in such conditions.
Choice D reason: Glucocorticoids increase, not lower, blood glucose by promoting gluconeogenesis and insulin resistance. Physiologic doses maintain normal glucose metabolism in adrenal insufficiency but do not replace insulin’s role. This statement is inaccurate, as glucocorticoids oppose insulin’s glucose-lowering effects, even at physiologic levels.
A client with type 1 diabetes is receiving insulin therapy. The nurse observes the patient experiencing symptoms such as sweating, trembling, and confusion. What is the nurse’s first action?
A. Administer simple, fast-acting carbohydrates to the client
Sweating, trembling, and confusion indicate hypoglycemia in type 1 diabetes, likely from excess insulin. Administering fast-acting carbohydrates (e.g., glucose tablets, juice) rapidly raises blood glucose by providing readily absorbable sugars, reversing neuroglycopenic symptoms. This is the first action to prevent seizures or coma, ensuring immediate stabilization.
B. Administer the prescribed dose of insulin since it was not yet given
Administering insulin during hypoglycemia would further lower blood glucose, exacerbating symptoms and risking severe outcomes like unconsciousness. Insulin drives glucose into cells, worsening the deficit. This action is contraindicated and dangerous, as it directly opposes the need to raise blood sugar immediately.
C. Order the client a meal with complex carbohydrates and proteins
Complex carbohydrates and proteins digest slowly, providing delayed glucose release, unsuitable for acute hypoglycemia requiring rapid correction. While appropriate for long-term glucose stability, this is not the first action, as it fails to address the urgent need for fast-acting sugars to reverse symptoms.
D. Call the healthcare provider to change the prescription for insulin
Calling the healthcare provider delays treatment of hypoglycemia, which requires immediate carbohydrate administration to prevent neurological damage. While provider consultation may follow for insulin adjustment, it is not the first action. This choice is inappropriate, as it postpones critical intervention needed for symptom resolution.
Full Explanation
Choice A reason: Sweating, trembling, and confusion indicate hypoglycemia in type 1 diabetes, likely from excess insulin. Administering fast-acting carbohydrates (e.g., glucose tablets, juice) rapidly raises blood glucose by providing readily absorbable sugars, reversing neuroglycopenic symptoms. This is the first action to prevent seizures or coma, ensuring immediate stabilization.
Choice B reason: Administering insulin during hypoglycemia would further lower blood glucose, exacerbating symptoms and risking severe outcomes like unconsciousness. Insulin drives glucose into cells, worsening the deficit. This action is contraindicated and dangerous, as it directly opposes the need to raise blood sugar immediately.
Choice C reason: Complex carbohydrates and proteins digest slowly, providing delayed glucose release, unsuitable for acute hypoglycemia requiring rapid correction. While appropriate for long-term glucose stability, this is not the first action, as it fails to address the urgent need for fast-acting sugars to reverse symptoms.
Choice D reason: Calling the healthcare provider delays treatment of hypoglycemia, which requires immediate carbohydrate administration to prevent neurological damage. While provider consultation may follow for insulin adjustment, it is not the first action. This choice is inappropriate, as it postpones critical intervention needed for symptom resolution.
A client with chronic kidney disease has a low red blood cell count. What is the best response by the nurse?
A. Your kidneys are allowing a lot of red blood cells to leak out in your urine and causing you to become anemic
Chronic kidney disease (CKD) does not primarily cause anemia by leaking red blood cells (RBCs) into urine. While hematuria may occur in some renal conditions, anemia in CKD results mainly from reduced erythropoietin production, not RBC loss. This statement is inaccurate, as it misrepresents the primary mechanism of anemia in CKD.
B. The inflammatory process attacking your kidneys is also attacking your red blood cells
Inflammation in CKD may contribute to anemia by suppressing erythropoiesis through cytokine release, but it does not directly attack RBCs. The primary cause is erythropoietin deficiency due to impaired renal function. This statement is inaccurate, as it overstates inflammation’s role and ignores the key hormonal mechanism in CKD-related anemia.
C. The high pressure in your vessels that caused damage to your kidneys is also causing your red blood cells to burst
High vascular pressure in CKD can damage kidneys but does not directly cause RBCs to burst (hemolysis). Anemia in CKD stems from reduced erythropoietin, not mechanical RBC destruction. This statement is inaccurate, as it incorrectly links hypertension’s renal effects to direct RBC damage, misrepresenting the anemia’s cause.
D. Your kidneys are not able to synthesize the hormone responsible for stimulating red blood cell production
CKD causes anemia due to reduced erythropoietin synthesis by damaged kidneys. Erythropoietin stimulates RBC production in bone marrow. In CKD, impaired renal function decreases erythropoietin, leading to anemia. This statement is accurate, as it correctly identifies the hormonal deficiency as the primary cause of low RBC counts in CKD.
Full Explanation
Choice A reason: Chronic kidney disease (CKD) does not primarily cause anemia by leaking red blood cells (RBCs) into urine. While hematuria may occur in some renal conditions, anemia in CKD results mainly from reduced erythropoietin production, not RBC loss. This statement is inaccurate, as it misrepresents the primary mechanism of anemia in CKD.
Choice B reason: Inflammation in CKD may contribute to anemia by suppressing erythropoiesis through cytokine release, but it does not directly attack RBCs. The primary cause is erythropoietin deficiency due to impaired renal function. This statement is inaccurate, as it overstates inflammation’s role and ignores the key hormonal mechanism in CKD-related anemia.
Choice C reason: High vascular pressure in CKD can damage kidneys but does not directly cause RBCs to burst (hemolysis). Anemia in CKD stems from reduced erythropoietin, not mechanical RBC destruction. This statement is inaccurate, as it incorrectly links hypertension’s renal effects to direct RBC damage, misrepresenting the anemia’s cause.
Choice D reason: CKD causes anemia due to reduced erythropoietin synthesis by damaged kidneys. Erythropoietin stimulates RBC production in bone marrow. In CKD, impaired renal function decreases erythropoietin, leading to anemia. This statement is accurate, as it correctly identifies the hormonal deficiency as the primary cause of low RBC counts in CKD.