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NurseDive Free Nursing Practice Question
A nurse is reviewing laboratory results for a client who has pericarditis. Which of the following blood test results should the nurse anticipate? (Select All that Apply.)
A. Elevated erythrocyte sedimentation rate (ESR)
Elevated erythrocyte sedimentation rate (ESR): Elevated ESR is commonly seen in inflammatory conditions such as pericarditis. ESR measures the rate at which red blood cells settle in a tube of blood over a certain period, and elevated levels indicate inflammation or tissue damage.
B. Increased C-reactive protein (CRP)
Increased C-reactive protein (CRP): CRP is an acute-phase reactant produced by the liver in response to inflammation. Elevated CRP levels are indicative of inflammation, making it a useful marker in pericarditis.
C. Elevated thyroid-stimulating hormone (TSH)
Elevated thyroid-stimulating hormone (TSH): Elevated TSH levels are not typically associated with pericarditis. TSH is a hormone produced by the pituitary gland that stimulates the thyroid gland to produce thyroid hormones. Elevated TSH levels are seen in conditions such as hypothyroidism.
D. Increased brain natriuretic peptide (BNP)
Increased brain natriuretic peptide (BNP): BNP is a hormone produced by the heart in response to increased pressure and volume overload. Pericarditis can lead to increased pressure within the heart, resulting in elevated BNP levels. Therefore, increased BNP levels are anticipated in pericarditis.
E. Increased troponin I
Increased troponin I: Troponin I is a cardiac biomarker released into the bloodstream when there is damage to cardiac muscle cells. While pericarditis primarily involves inflammation of the pericardium (the sac surrounding the heart), severe cases can lead to myocardial involvement and subsequent release of troponin I. Therefore, increased troponin I levels may be observed in pericarditis, especially if there is myocardial involvement.
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Full Explanation
A. Elevated erythrocyte sedimentation rate (ESR): Elevated ESR is commonly seen in inflammatory conditions such as pericarditis. ESR measures the rate at which red blood cells settle in a tube of blood over a certain period, and elevated levels indicate inflammation or tissue damage.
B. Increased C-reactive protein (CRP): CRP is an acute-phase reactant produced by the liver in response to inflammation. Elevated CRP levels are indicative of inflammation, making it a useful marker in pericarditis.
C. Elevated thyroid-stimulating hormone (TSH): Elevated TSH levels are not typically associated with pericarditis. TSH is a hormone produced by the pituitary gland that stimulates the thyroid gland to produce thyroid hormones. Elevated TSH levels are seen in conditions such as hypothyroidism.
D. Increased brain natriuretic peptide (BNP): BNP is a hormone produced by the heart in response to increased pressure and volume overload. Pericarditis can lead to increased pressure within the heart, resulting in elevated BNP levels. Therefore, increased BNP levels are anticipated in pericarditis.
E. Increased troponin I: Troponin I is a cardiac biomarker released into the bloodstream when there is damage to cardiac muscle cells. While pericarditis primarily involves inflammation of the pericardium (the sac surrounding the heart), severe cases can lead to myocardial involvement and subsequent release of troponin I. Therefore, increased troponin I levels may be observed in pericarditis, especially if there is myocardial involvement.
Similar Questions
A nurse is teaching a client who has restrictive cardiomyopathy (RCM). Which of the following statements should the nurse include in the client teaching to explain the cause of RCM?
A. "Your heart condition is caused by excessive stretching of the ventricles."
"Your heart condition is caused by excessive stretching of the ventricles": This statement is incorrect. Excessive stretching of the ventricles typically occurs in dilated cardiomyopathy, not restrictive cardiomyopathy (RCM). In RCM, the primary issue is not excessive stretching but rather stiffening of the ventricular walls.
B. "Your heart condition is caused from stiffening of the walls of the ventricles."
"Your heart condition is caused from stiffening of the walls of the ventricles": This statement is correct. Restrictive cardiomyopathy (RCM) is characterized by abnormal stiffening (fibrosis) of the ventricular walls, which impairs the heart's ability to fill properly during the diastolic phase of the cardiac cycle. This stiffening restricts the heart's ability to relax and fill with blood efficiently.
C. "Your heart condition is caused by thickening of the ventricular walls and septum."
"Your heart condition is caused by thickening of the ventricular walls and septum": This statement describes hypertrophic cardiomyopathy (HCM), not restrictive cardiomyopathy (RCM). In HCM, there is abnormal thickening of the ventricular walls and septum, leading to impaired filling of the ventricles and potential obstruction of blood flow out of the heart.
D. "Your heart condition is caused when the ventricular tissue becomes fibrous and fatty."
"Your heart condition is caused when the ventricular tissue becomes fibrous and fatty": This statement describes arrhythmogenic right ventricular cardiomyopathy (ARVC), not restrictive cardiomyopathy (RCM). ARVC is characterized by replacement of myocardial tissue with fibrous and fatty tissue, primarily affecting the right ventricle.
Full Explanation
A. "Your heart condition is caused by excessive stretching of the ventricles": This statement is incorrect. Excessive stretching of the ventricles typically occurs in dilated cardiomyopathy, not restrictive cardiomyopathy (RCM). In RCM, the primary issue is not excessive stretching but rather stiffening of the ventricular walls.
B. "Your heart condition is caused from stiffening of the walls of the ventricles": This statement is correct. Restrictive cardiomyopathy (RCM) is characterized by abnormal stiffening (fibrosis) of the ventricular walls, which impairs the heart's ability to fill properly during the diastolic phase of the cardiac cycle. This stiffening restricts the heart's ability to relax and fill with blood efficiently.
C. "Your heart condition is caused by thickening of the ventricular walls and septum": This statement describes hypertrophic cardiomyopathy (HCM), not restrictive cardiomyopathy (RCM). In HCM, there is abnormal thickening of the ventricular walls and septum, leading to impaired filling of the ventricles and potential obstruction of blood flow out of the heart.
D. "Your heart condition is caused when the ventricular tissue becomes fibrous and fatty": This statement describes arrhythmogenic right ventricular cardiomyopathy (ARVC), not restrictive cardiomyopathy (RCM). ARVC is characterized by replacement of myocardial tissue with fibrous and fatty tissue, primarily affecting the right ventricle.
A nurse is planning care for a client who has peripheral venous disease. Which of the following interventions should the nurse include in the plan of care? (Select All that Apply.)
A. Intermittent pneumatic compression pumps
Intermittent pneumatic compression pumps: Intermittent pneumatic compression pumps can help improve circulation in the lower extremities by assisting with venous return. These devices inflate and deflate sequentially, promoting venous blood flow and reducing edema in clients with peripheral venous disease.
B. Ankle-brachial index test
Ankle-brachial index test: The ankle-brachial index (ABI) test is a non-invasive vascular study used to assess peripheral arterial disease (PAD). It compares the blood pressure in the ankle with the blood pressure in the arm to evaluate arterial circulation.
C. Layered wraps
Layered wraps: Layered wraps, such as compression bandages or stockings, are typically used in the management of venous insufficiency and venous ulcers. They are not as commonly utilized in peripheral venous disease, however, they may be beneficial for some clients with PVD.
D. Elevation of legs
Elevation of legs: Elevating the legs above the level of the heart can help reduce edema and improve venous return in clients with peripheral venous disease. Gravity assists in draining blood from the lower extremities back toward the heart, thereby reducing swelling and discomfort.
E. Exercise
Exercise: Regular physical activity, particularly lower extremity exercises such as walking, can promote venous return and improve circulation in clients with peripheral venous disease. Exercise helps to pump blood back to the heart, reducing venous stasis and the risk of complications such as venous thrombosis.
Full Explanation
A. Intermittent pneumatic compression pumps: Intermittent pneumatic compression pumps can help improve circulation in the lower extremities by assisting with venous return. These devices inflate and deflate sequentially, promoting venous blood flow and reducing edema in clients with peripheral venous disease.
B. Ankle-brachial index test: The ankle-brachial index (ABI) test is a non-invasive vascular study used to assess peripheral arterial disease (PAD). It compares the blood pressure in the ankle with the blood pressure in the arm to evaluate arterial circulation.
C. Layered wraps: Layered wraps, such as compression bandages or stockings, are typically used in the management of venous insufficiency and venous ulcers. They are not as commonly utilized in peripheral venous disease, however, they may be beneficial for some clients with PVD.
D. Elevation of legs: Elevating the legs above the level of the heart can help reduce edema and improve venous return in clients with peripheral venous disease. Gravity assists in draining blood from the lower extremities back toward the heart, thereby reducing swelling and discomfort.
E. Exercise: Regular physical activity, particularly lower extremity exercises such as walking, can promote venous return and improve circulation in clients with peripheral venous disease. Exercise helps to pump blood back to the heart, reducing venous stasis and the risk of complications such as venous thrombosis.
A nurse is providing teaching to a client about recognizing manifestations of an impending cardiac arrest. Which of the following manifestations should the nurse include in the teaching?
A. Muscular aches in the leg
Muscular aches in the leg: Muscular aches in the leg are not typically indicative of an impending cardiac arrest. While leg pain or cramping can be associated with peripheral vascular disease or venous insufficiency, they are not specific signs of cardiac arrest.
B. Profound fatigue
Profound fatigue: Profound fatigue can be a warning sign of an impending cardiac arrest. Fatigue or weakness can result from inadequate blood flow to the heart muscle, which may occur prior to a cardiac event. Additionally, systemic effects of cardiovascular compromise can lead to generalized weakness and fatigue.
C. Severe headache
Severe headache: While severe headache can be associated with conditions such as hypertension or intracranial bleeding, it is not a typical manifestation of an impending cardiac arrest. Headaches may occur as a result of stress or anxiety related to the cardiac event, but they are not a direct warning sign of impending cardiac arrest.
D. Ringing in the ears
Ringing in the ears: Ringing in the ears, also known as tinnitus, is not typically associated with an impending cardiac arrest. Tinnitus can result from various factors such as noise exposure, ear infections, or certain medications, but it is not considered a warning sign of impending cardiac arrest.
Full Explanation
A. Muscular aches in the leg: Muscular aches in the leg are not typically indicative of an impending cardiac arrest. While leg pain or cramping can be associated with peripheral vascular disease or venous insufficiency, they are not specific signs of cardiac arrest.
B. Profound fatigue: Profound fatigue can be a warning sign of an impending cardiac arrest. Fatigue or weakness can result from inadequate blood flow to the heart muscle, which may occur prior to a cardiac event. Additionally, systemic effects of cardiovascular compromise can lead to generalized weakness and fatigue.
C. Severe headache: While severe headache can be associated with conditions such as hypertension or intracranial bleeding, it is not a typical manifestation of an impending cardiac arrest. Headaches may occur as a result of stress or anxiety related to the cardiac event, but they are not a direct warning sign of impending cardiac arrest.
D. Ringing in the ears: Ringing in the ears, also known as tinnitus, is not typically associated with an impending cardiac arrest. Tinnitus can result from various factors such as noise exposure, ear infections, or certain medications, but it is not considered a warning sign of impending cardiac arrest.