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What type of shock is characterized by a hypersensitivity reaction causing massive vasodilation and capillary leaks, which may occur with a drug or latex allergy?

A. Anaphylactic shock.

Anaphylactic shock is the correct answer. Anaphylactic shock is a severe and potentially life-threatening allergic reaction that causes massive vasodilation, capillary leakage, and bronchoconstriction. It can occur rapidly after exposure to an allergen such as certain foods, insect stings, medications, or latex. The release of histamine and other inflammatory mediators leads to systemic vasodilation and increased permeability of blood vessels, resulting in a sudden drop in blood pressure and compromised organ perfusion.

B. Cardiogenic shock.

Cardiogenic shock is characterized by inadequate cardiac output due to severe heart dysfunction. It is typically caused by conditions like myocardial infarction, heart failure, or other cardiac issues. While allergic reactions can potentially affect the cardiovascular system, the symptoms described in the question are more indicative of anaphylactic shock.

C. Neurogenic shock.

Neurogenic shock results from disruption of sympathetic nervous system control over blood vessel tone, often due to spinal cord injury or severe emotional distress. This leads to widespread vasodilation and a sudden drop in blood pressure. While it can cause hemodynamic instability, it doesn't specifically involve the hypersensitivity reaction and capillary leaks associated with anaphylactic shock.

D. Hypovolemic shock.

Hypovolemic shock is caused by a significant loss of blood volume or bodily fluids, leading to decreased intravascular volume and subsequent inadequate tissue perfusion. This shock type is not directly related to a hypersensitivity reaction or capillary leakage caused by allergic responses. 2 / 2

This question is an excerpt from Nurse Dive's nursing test bank - Pediatrics Midterm V1 2023 Test 3 Proctored Exam. Take the full exam now


Full Explanation

The correct answer is choice A: Anaphylactic shock.

Choice A rationale:

Anaphylactic shock is the correct answer. Anaphylactic shock is a severe and potentially life-threatening allergic reaction that causes massive vasodilation, capillary leakage, and bronchoconstriction. It can occur rapidly after exposure to an allergen such as certain foods, insect stings, medications, or latex. The release of histamine and other inflammatory mediators leads to systemic vasodilation and increased permeability of blood vessels, resulting in a sudden drop in blood pressure and compromised organ perfusion.

Choice B rationale:

Cardiogenic shock is characterized by inadequate cardiac output due to severe heart dysfunction. It is typically caused by conditions like myocardial infarction, heart failure, or other cardiac issues. While allergic reactions can potentially affect the cardiovascular system, the symptoms described in the question are more indicative of anaphylactic shock.

Choice C rationale:

Neurogenic shock results from disruption of sympathetic nervous system control over blood vessel tone, often due to spinal cord injury or severe emotional distress. This leads to widespread vasodilation and a sudden drop in blood pressure. While it can cause hemodynamic instability, it doesn't specifically involve the hypersensitivity reaction and capillary leaks associated with anaphylactic shock.

Choice D rationale:

Hypovolemic shock is caused by a significant loss of blood volume or bodily fluids, leading to decreased intravascular volume and subsequent inadequate tissue perfusion. This shock type is not directly related to a hypersensitivity reaction or capillary leakage caused by allergic responses. 2 / 2


Similar Questions

QUESTION

What is a common sign of digoxin toxicity?

A. Vomiting.

Vomiting is a common sign of digoxin toxicity. It occurs due to the drug's effects on the gastrointestinal system, which can lead to nausea and vomiting as the body attempts to expel the toxin. This symptom is particularly significant as it can indicate elevated digoxin levels that may require medical intervention.

B. Tachycardia.

Tachycardia, or an increased heart rate, can occur with digoxin toxicity; however, it is more commonly associated with inadequate therapeutic levels rather than toxicity itself. Digoxin usually causes bradycardia (a slower heart rate) when at therapeutic levels, making tachycardia less indicative of toxicity.

C. Bradypnea.

Bradypnea (slow breathing) is not a common sign of digoxin toxicity. While digoxin primarily affects the heart, it doesn't typically have a direct impact on respiratory function.

D. Seizures.

Seizures are not a common sign of digoxin toxicity. Digoxin toxicity primarily affects the heart's electrical activity and can lead to arrhythmias, but it doesn't typically cause seizures.

Full Explanation

The correct answer is Choice A.Choice A rationale: Vomiting is a common sign of digoxin toxicity. It occurs due to the drug's effects on the gastrointestinal system, which can lead to nausea and vomiting as the body attempts to expel the toxin. This symptom is particularly significant as it can indicate elevated digoxin levels that may require medical intervention.Choice B rationale: Tachycardia, or an increased heart rate, can occur with digoxin toxicity; however, it is more commonly associated with inadequate therapeutic levels rather than toxicity itself. Digoxin usually causes bradycardia (a slower heart rate) when at therapeutic levels, making tachycardia less indicative of toxicity.Choice C rationale: Bradypnea, or slow breathing, is not a typical sign of digoxin toxicity. While respiratory issues can arise from various conditions, they are not specifically linked to digoxin levels. Monitoring respiratory rate is essential in clinical settings but does not directly correlate with digoxin toxicity.Choice D rationale: Seizures are not a common sign of digoxin toxicity. While severe cases might lead to neurological symptoms due to electrolyte imbalances or other complications, seizures are not typically associated with digoxin overdose. Instead, they may suggest other underlying issues requiring evaluation.

QUESTION

What is a common clinical manifestation of juvenile hypothyroidism?

A. Diarrhea.

Diarrhea is not a common clinical manifestation of juvenile hypothyroidism. Hypothyroidism is characterized by an underactive thyroid gland, leading to a decrease in metabolic activity. Symptoms of hypothyroidism include fatigue, weight gain, cold intolerance, constipation, and dry skin, but not diarrhea.

B. Dry skin.

Dry skin is a common clinical manifestation of juvenile hypothyroidism. The decreased thyroid function results in a slower metabolic rate, which can lead to dry and coarse skin due to reduced oil production. This symptom is often seen in hypothyroid patients.

C. Insomnia.

Insomnia is not a common clinical manifestation of juvenile hypothyroidism. Hypothyroidism usually leads to fatigue and excessive sleepiness rather than insomnia.

D. Accelerated growth.

Accelerated growth is not a common clinical manifestation of juvenile hypothyroidism. In fact, the opposite is true. Hypothyroidism in children can lead to growth retardation due to the decreased metabolic rate and altered hormonal balance.

Full Explanation

The correct answer is Choice B: Dry skin.

Choice A rationale:

Diarrhea is not a common clinical manifestation of juvenile hypothyroidism. Hypothyroidism is characterized by an underactive thyroid gland, leading to a decrease in metabolic activity. Symptoms of hypothyroidism include fatigue, weight gain, cold intolerance, constipation, and dry skin, but not diarrhea.

Choice B rationale:

Dry skin is a common clinical manifestation of juvenile hypothyroidism. The decreased thyroid function results in a slower metabolic rate, which can lead to dry and coarse skin due to reduced oil production. This symptom is often seen in hypothyroid patients.

Choice C rationale:

Insomnia is not a common clinical manifestation of juvenile hypothyroidism. Hypothyroidism usually leads to fatigue and excessive sleepiness rather than insomnia.

Choice D rationale:

Accelerated growth is not a common clinical manifestation of juvenile hypothyroidism. In fact, the opposite is true. Hypothyroidism in children can lead to growth retardation due to the decreased metabolic rate and altered hormonal balance.

QUESTION

Therapeutic management of most children with Hirschsprung's disease is primarily

A. surgical removal of the affected section of bowel.

Surgical removal of the affected section of bowel is the primary therapeutic management for most children with Hirschsprung's disease. Hirschsprung's disease is a congenital condition where there is a lack of nerve cells in a segment of the colon, leading to obstruction and dilation of the bowel. Surgical resection of the affected segment and reconnection of healthy bowel portions is the standard treatment to alleviate the obstruction and restore normal bowel function.

B. daily enemas.

Daily enemas might be used in some cases to manage symptoms temporarily, but they are not the primary therapeutic management for Hirschsprung's disease. Surgery is the mainstay of treatment.

C. permanent colostomy.

Permanent colostomy might be considered if the affected portion of bowel is extensive and cannot be safely reconnected after resection, but it's not the primary option for most children with Hirschsprung's disease.

D. low-fiber diet.

A low-fiber diet is not a therapeutic management for Hirschsprung's disease. Surgical intervention is necessary to address the underlying cause of obstruction, and diet modifications alone wouldn't resolve the condition.

Full Explanation

The correct answer is Choice A: Surgical removal of the affected section of bowel.

Choice A rationale:

Surgical removal of the affected section of bowel is the primary therapeutic management for most children with Hirschsprung's disease. Hirschsprung's disease is a congenital condition where there is a lack of nerve cells in a segment of the colon, leading to obstruction and dilation of the bowel. Surgical resection of the affected segment and reconnection of healthy bowel portions is the standard treatment to alleviate the obstruction and restore normal bowel function.

Choice B rationale:

Daily enemas might be used in some cases to manage symptoms temporarily, but they are not the primary therapeutic management for Hirschsprung's disease. Surgery is the mainstay of treatment.

Choice C rationale:

Permanent colostomy might be considered if the affected portion of bowel is extensive and cannot be safely reconnected after resection, but it's not the primary option for most children with Hirschsprung's disease.

Choice D rationale:

A low-fiber diet is not a therapeutic management for Hirschsprung's disease. Surgical intervention is necessary to address the underlying cause of obstruction, and diet modifications alone wouldn't resolve the condition.