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A woman with severe preeclampsia is being treated with bed rest and intravenous magnesium sulfate. The drug classification of this medication is

A. Antihypertensive

Choice A) Antihypertensive: This is not the correct classification of magnesium sulfate. Antihypertensives are drugs that lower blood pressure, such as beta blockers, calcium channel blockers, or angiotensin-converting enzyme inhibitors. Magnesium sulfate does not have a significant effect on blood pressure, and it is not used as a primary treatment for hypertension in preeclampsia.

B. Anticonvulsant

Choice B) Anticonvulsant: This is the correct classification of magnesium sulfate. Anticonvulsants are drugs that prevent or reduce the frequency and severity of seizures, such as phenytoin, valproic acid, or carbamazepine. Magnesium sulfate is used as a prophylactic and therapeutic agent for eclampsia, which is a life-threatening complication of preeclampsia characterized by seizures. Magnesium sulfate acts by blocking the neuromuscular transmission and reducing the cerebral edema and vasospasm.

C. Tocolytic

Choice C) Tocolytic: This is not the correct classification of magnesium sulfate. Tocolytics are drugs that inhibit uterine contractions and delay preterm labor, such as terbutaline, nifedipine, or indomethacin. Magnesium sulfate is not effective as a tocolytic agent, and it is not recommended for this purpose by the American College of Obstetricians and Gynecologists.

D. Diuretic

Choice D) Diuretic: This is not the correct classification of magnesium sulfate. Diuretics are drugs that increase urine output and reduce fluid retention, such as furosemide, hydrochlorothiazide, or spironolactone. Magnesium sulfate does not have a diuretic effect, and it can cause fluid overload and pulmonary edema if administered in excess.

This question is an excerpt from Nurse Dive's nursing test bank - ATI Maternity Proctored Exam 2. Take the full exam now


Full Explanation

Choice A) Antihypertensive: This is not the correct classification of magnesium sulfate. Antihypertensives are drugs that lower blood pressure, such as beta blockers, calcium channel blockers, or angiotensin-converting enzyme inhibitors. Magnesium sulfate does not have a significant effect on blood pressure, and it is not used as a primary treatment for hypertension in preeclampsia.

Choice B) Anticonvulsant: This is the correct classification of magnesium sulfate. Anticonvulsants are drugs that prevent or reduce the frequency and severity of seizures, such as phenytoin, valproic acid, or carbamazepine.

Magnesium sulfate is used as a prophylactic and therapeutic agent for eclampsia, which is a life-threatening complication of preeclampsia characterized by seizures. Magnesium sulfate acts by blocking the neuromuscular transmission and reducing the cerebral edema and vasospasm.

Choice C) Tocolytic: This is not the correct classification of magnesium sulfate. Tocolytics are drugs that inhibit uterine contractions and delay preterm labor, such as terbutaline, nifedipine, or indomethacin. Magnesium sulfate is not effective as a tocolytic agent, and it is not recommended for this purpose by the American College of Obstetricians and Gynecologists.

Choice D) Diuretic: This is not the correct classification of magnesium sulfate. Diuretics are drugs that increase urine output and reduce fluid retention, such as furosemide, hydrochlorothiazide, or spironolactone. Magnesium sulfate does not have a diuretic effect, and it can cause fluid overload and pulmonary edema if administered in excess.


Similar Questions

QUESTION

Which of the following would the nurse expect to assess in a newborn who develops sepsis?

A. Increased urinary output

Choice A) Increased urinary output: This is not a sign of sepsis in newborns. In fact, sepsis can cause reduced urinary output due to poor blood flow to the kidneys and dehydration.

B. Hypothermia

Choice B) Hypothermia: This is a sign of sepsis in newborns. Sepsis can cause changes in temperature, often fever, but sometimes low temperature. Hypothermia can indicate a severe infection that affects the body's ability to regulate its temperature.

C. Wakefulness

Choice C) Wakefulness: This is not a sign of sepsis in newborns. Sepsis can cause reduced activity and lethargy due to inflammation and organ dysfunction.

D. Interest in feeding

Choice D) Interest in feeding: This is not a sign of sepsis in newborns. Sepsis can cause reduced sucking and difficulty feeding due to poor appetite, nausea, vomiting, and abdominal distension.

Full Explanation

Choice A) Increased urinary output: This is not a sign of sepsis in newborns. In fact, sepsis can cause reduced urinary output due to poor blood flow to the kidneys and dehydration.

Choice B) Hypothermia: This is a sign of sepsis in newborns. Sepsis can cause changes in temperature, often fever, but sometimes low temperature. Hypothermia can indicate a severe infection that affects the body's ability to regulate its temperature.

Choice C) Wakefulness: This is not a sign of sepsis in newborns. Sepsis can cause reduced activity and lethargy due to inflammation and organ dysfunction.

Choice D) Interest in feeding: This is not a sign of sepsis in newborns. Sepsis can cause reduced sucking and difficulty feeding due to poor appetite, nausea, vomiting, and abdominal distension.

QUESTION

Methotrexate is recommended as part of the treatment plan for which obstetric complication?

A. Missed abortion

Choice A) Missed abortion: This is not the correct obstetric complication for methotrexate treatment. A missed abortion is a type of miscarriage in which the fetus has died but the products of conception are still retained in the uterus. Methotrexate is not used for this condition, as it can cause toxicity and bleeding. The usual treatment options for a missed abortion are expectant management, medical induction, or surgical evacuation.

B. Abruptio placentae

Choice B) Abruptio placentae: This is not the correct obstetric complication for methotrexate treatment. Abruptio placentae is a condition in which the placenta separates from the uterine wall before delivery, causing bleeding and fetal distress. Methotrexate is not used for this condition, as it can worsen the bleeding and harm the fetus. The usual treatment options for abruptio placentae depend on the severity of the condition and the gestational age, but they may include fluid resuscitation, blood transfusion, tocolysis, or emergency delivery.

C. Unruptured ectopic pregnancy

Choice C) Unruptured ectopic pregnancy: This is the correct obstetric complication for methotrexate treatment. An ectopic pregnancy is a pregnancy that implants outside of the uterine cavity, usually in the fallopian tube. An unruptured ectopic pregnancy is one that has not caused any bleeding or rupture of the tube. Methotrexate is used for this condition, as it can dissolve the pregnancy tissue and prevent further growth and complications. Methotrexate is given as an injection and works by inhibiting folic acid metabolism, which is essential for cell division. Methotrexate is only suitable for patients who have stable vital signs, low levels of human chorionic gonadotropin (hCG), and no fetal heartbeat or cardiac activity detected by ultrasound.

D. Complete hydatidiform mole

Choice D) Complete hydatidiform mole: This is not the correct obstetric complication for methotrexate treatment. A complete hydatidiform mole is a type of gestational trophoblastic disease in which there is an abnormal proliferation of placental tissue without any fetal development. Methotrexate is not used for this condition, as it can cause resistance and recurrence. The usual treatment option for a complete hydatidiform mole is suction curettage, which removes the molar tissue from the uterus.

Full Explanation

Choice A) Missed abortion: This is not the correct obstetric complication for methotrexate treatment. A missed abortion is a type of miscarriage in which the fetus has died but the products of conception are still retained in the uterus. Methotrexate is not used for this condition, as it can cause toxicity and bleeding. The usual treatment options for a missed abortion are expectant management, medical induction, or surgical evacuation.

Choice B) Abruptio placentae: This is not the correct obstetric complication for methotrexate treatment. Abruptio placentae is a condition in which the placenta separates from the uterine wall before delivery, causing bleeding and fetal distress. Methotrexate is not used for this condition, as it can worsen the bleeding and harm the fetus. The usual treatment options for abruptio placentae depend on the severity of the condition and the gestational age, but they may include fluid resuscitation, blood transfusion, tocolysis, or emergency delivery.

Choice C) Unruptured ectopic pregnancy: This is the correct obstetric complication for methotrexate treatment. An ectopic pregnancy is a pregnancy that implants outside of the uterine cavity, usually in the fallopian tube. An unruptured ectopic pregnancy is one that has not caused any bleeding or rupture of the tube. Methotrexate is used for this condition, as it can dissolve the pregnancy tissue and prevent further growth and complications.

Methotrexate is given as an injection and works by inhibiting folic acid metabolism, which is essential for cell division.

Methotrexate is only suitable for patients who have stable vital signs, low levels of human chorionic gonadotropin (hCG), and no fetal heartbeat or cardiac activity detected by ultrasound.

Choice D) Complete hydatidiform mole: This is not the correct obstetric complication for methotrexate treatment. A complete hydatidiform mole is a type of gestational trophoblastic disease in which there is an abnormal proliferation of placental tissue without any fetal development. Methotrexate is not used for this condition, as it can cause resistance and recurrence. The usual treatment option for a complete hydatidiform mole is suction curettage, which removes the molar tissue from the uterus.

QUESTION

Decreased surfactant production in the preterm lung is a problem because surfactant

A. dilates the bronchioles, decreasing airway resistance.

Choice A) dilates the bronchioles, decreasing airway resistance: This is not the correct function of surfactant. Surfactant is a substance that reduces the surface tension of the fluid that lines the alveoli, which are the tiny air sacs in the lungs where gas exchange occurs. Surfactant does not affect the diameter of the bronchioles, which are the small airways that branch from the bronchi. Bronchodilation and bronchoconstriction are regulated by the autonomic nervous system and various mediators, such as histamine, epinephrine, and acetylcholine.

B. provides transportation for oxygen to enter the blood supply.

Choice B) provides transportation for oxygen to enter the blood supply: This is not the correct function of surfactant. Surfactant does not transport oxygen or any other gas. Oxygen diffuses from the alveoli into the capillaries, where it binds to hemoglobin in the red blood cells. The red blood cells then transport oxygen to the tissues via the blood circulation. Surfactant does not play a role in this process.

C. keeps the alveoli open during expiration.

Choice C) keeps the alveoli open during expiration: This is the correct function of surfactant. Surfactant prevents the alveoli from collapsing during expiration by lowering the surface tension of the fluid that lines them. This allows for easier breathing and better gas exchange. Without enough surfactant, the alveoli tend to collapse and stick together, causing atelectasis, which is a condition where some or all of a lung collapses. Atelectasis can lead to hypoxia, respiratory distress, and infection.

D. causes increased permeability of the alveoli.

Choice D) causes increased permeability of the alveoli: This is not the correct function of surfactant. Surfactant does not increase or decrease the permeability of the alveoli, which is the ability of substances to pass through them. Permeability of the alveoli depends on several factors, such as pressure gradients, solubility, molecular size, and membrane thickness. Increased permeability of the alveoli can occur in conditions such as acute respiratory distress syndrome (ARDS), where fluid leaks into the alveolar space and impairs gas exchange. Surfactant does not cause this condition, but it can be affected by it.

Full Explanation

Choice A) dilates the bronchioles, decreasing airway resistance: This is not the correct function of surfactant.

Surfactant is a substance that reduces the surface tension of the fluid that lines the alveoli, which are the tiny air sacs in the lungs where gas exchange occurs. Surfactant does not affect the diameter of the bronchioles, which are the small airways that branch from the bronchi. Bronchodilation and bronchoconstriction are regulated by the autonomic nervous system and various mediators, such as histamine, epinephrine, and acetylcholine.

Choice B) provides transportation for oxygen to enter the blood supply: This is not the correct function of surfactant. Surfactant does not transport oxygen or any other gas. Oxygen diffuses from the alveoli into the capillaries, where it binds to hemoglobin in the red blood cells. The red blood cells then transport oxygen to the tissues via the blood circulation. Surfactant does not play a role in this process.

Choice C) keeps the alveoli open during expiration: This is the correct function of surfactant. Surfactant prevents the alveoli from collapsing during expiration by lowering the surface tension of the fluid that lines them. This allows for easier breathing and better gas exchange. Without enough surfactant, the alveoli tend to collapse and stick together, causing atelectasis, which is a condition where some or all of a lung collapses. Atelectasis can lead to hypoxia, respiratory distress, and infection.

Choice D) causes increased permeability of the alveoli: This is not the correct function of surfactant. Surfactant does not increase or decrease the permeability of the alveoli, which is the ability of substances to pass through them.

Permeability of the alveoli depends on several factors, such as pressure gradients, solubility, molecular size, and membrane thickness. Increased permeability of the alveoli can occur in conditions such as acute respiratory distress syndrome (ARDS), where fluid leaks into the alveolar space and impairs gas exchange. Surfactant does not cause this condition, but it can be affected by it.